By Andrew M. Churg, Jeffrey L. Myers, Henry D. Tazelaar, Joanne L. Wright
This revised variation of a cornerstone textbook and reference on pulmonary pathology accommodates the newest advances within the box, exhibiting how molecular biology povides a better realizing of lung improvement. It bargains new insights into the analysis of neoplastic and non-neoplastic lung disorder; presents pertinent details on scientific gains, epidemiology, and pathogenic mechanisms of lung sickness, and a lot more. Read more...
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159 VEGF expression is affected in a paracrine fashion by other protein mediators. 5,163–165 The pleural lymphatic plexus consists of lymphatic trunks located mainly at the junctions of the interlobular septa with the pleura, fed by fine lymphatic capillaries crossing the polygonal spaces enclosed by the larger lymphatic trunks. Lateral channels off the smaller lymphatics end blindly. The pleural lymphatics are larger in the lower zones of the lung than in the upper zones,163,164 presumably because the vertical gradient in hydrostatic pressure favors greater lymph formation at the bases.
Annu Rev Physiol 1991;53:375–385 92. Crouch E, Wright JR. Surfactant proteins A and D and pulmonary host defense. Annu Rev Physiol 2001;63:521–554 93. Kuroki Y, Mason RJ, Voelker DR. Alveolar type II cells express a high-affinity receptor for pulmonary surfactant protein A. Proc Natl Acad Sci U S A 1988;85:5566–5570 94. Kuan SF, Rust K, Crouch E.
However, it also induces expression of interstitial collagenase in endothelial cells, thus potentially affecting smooth muscle cell proliferation or matrix reorganization. 159 VEGF expression is affected in a paracrine fashion by other protein mediators. 5,163–165 The pleural lymphatic plexus consists of lymphatic trunks located mainly at the junctions of the interlobular septa with the pleura, fed by fine lymphatic capillaries crossing the polygonal spaces enclosed by the larger lymphatic trunks.