Dental Pathology: A Practical Introduction by Pieter Slootweg

By Pieter Slootweg

This new, up to date variation of Dental Pathology will help in the popularity and prognosis of gross dental abnormalities. subject matters lined comprise disturbances in teeth formation, obtained dental ailments, together with caries and its sequelae, periodontal sickness, and odontogenic tumours. essentially appropriate wisdom is equipped at the histology and histopathology of the alterations which are visible in illnesses of the dental and periodontal tissues, and a spotlight can also be paid to the histological adjustments brought on via dental therapy. in comparison with the 1st version, ca. 50% extra illustrations are integrated and additional info is supplied on diagnostically tough situations. This paintings should be a useful significant other for dental practitioners who intend to make diagnoses in such circumstances and also will be of worth for surgical pathologists, scientific geneticists, and oral and maxillofacial surgeons.

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Barron MJ, McDonnell ST, MacKie I, Dixon MJ (2008) Review. Hereditary dentine disorders: dentinogenesis imperfect and dentine dysplasia. Orphanet J Rare Dis 3:31 12. Kim JW, Simmer JP (2007) Hereditary dentin defects. J Dent Res 86:392–399 13. O’Carrol MK, Duncan WK, Perkins TM (1991) Dentin dysplasia: review of the literature and a proposed subclassification based on radiographic findings. Oral Surg Oral Med Oral Pathol 72:119–125 14. Parekh S, Kyriazidou A, Bloch-Zupan A et al (2006) Multiple pulp stones and shortened roots of unknown etiology.

Radiographically detectable abnormalities of the involved teeth include enlarged crowns, roots and pulps of primary molars; irregular resorption of primary molar roots; splayed roots of primary molars; and an abnormal medial position of the teeth [25]. Data on histology of the involved teeth are scarce, only one report mentioning them. These authors report the presence of tubular defects in the coronal dentin extending from the pulp towards the dentino-enamel junction. The circumpulpal dentin from the middle one-third of the root apically had an irregular tubular structure and lack a well-defined odontoblast layer (Fig.

In those cases, the teeth involved show bending and no angulation. This type of malformation is not included in the definition of dilaceration. Taurodontism In normal teeth, the pulp chamber shows a constriction at the level of the amelocemental junction. In taurodontism, the pulp chamber shows an increased vertical dimension, thus extending far into the root area of the involved tooth. As a consequence, in multirooted teeth, the splitting of the root into its several extensions, the so-called furcation, occurs at a level much more apically than is the case in normal 32 a 4 Disturbed Tooth Form c b Fig.

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